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By William Frederick. Koch

Dr. Koch’s study fascinated by the capacity to revive the body’s oxidation
mechanism again to its unique power, thereby re-equipping the physique with its innate skill to
restore and hold overall healthiness, not just in cancers but in addition in a number of its ‘allied diseases.’
This learn ended in Dr. Koch’s improvement of a number of man made antitoxins: Glyoxylide, Malonide and PBQ. those catalysts
became the stimulant essential to in achieving the oxidative separation of the ‘host cell/pathogen integration,’ while the pathogen
was an epidemic, a carcinogen, a bacterial toxin or an incompletely burned tissue metabolite. Dr. Koch effectively outlined the
position of the activated amine crew, the unfastened radical, the double bond and the Carbonyl team in pathogenesis and in its
correction.
Of old importance is the information that as early as 1919, Dr. Koch’s discoveries have been taking him in a course diametrically
opposed to the location held through prepared medication, which at the moment was once making an investment seriously within the improvement of radium and
surgery because the such a lot promising remedies for cancer.
After failing in its try and achieve sole keep an eye on over his study, equipped drugs introduced a fifty-year, limitless assault
aimed at discrediting Dr. Koch’s acceptance, clinical perform and study, in addition to these of any general practitioner who dared to
validate his Theories or use his Reagents. equipped medication constructed an intensive propaganda crusade, disseminated false
information on Reagent chemistry and publicly pushed aside the Koch Theories, which emphasised the connection between
environmental pollutants, nutritional deficiencies and a depleted oxidation mechanism, as basic initiators of the ailment process.
Because Dr. Koch persisted such vast persecution in regard to his technology, he decided that the medical/pharmacological
industry might perpetually stay unwilling to independently video display, record or validate any of his ongoing laboratory study or
medical case histories; hence because his loss of life, December nine, 1967, there were no real Koch Reagents reproduced. It
was as a result of the scurrilous intentions held via the medical/pharmacological that Dr. Koch intentionally
withheld particular wisdom required within the creation of practicable Koch Reagents. (Therefore, any claims to the contrary
should be seen as suspect.)

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J Infect Dis 2004;190: 468–476. Lazcano-Ponce E, Herrero R, Munoz N, Hernandez-Avila M, Salmerón J, Leyva A, Meijer C, Walboomers J: High prevalence of human papillomavirus infection in Mexican males: comparative study of penile-urethral swabs and urine samples. Sex Transm Dis 2001;28:277–280. Baldwin S, Wallace D, Papenfuss M, Abrahamsen M, Vaught L, Kornegay J, Hallum J, Redmond S, Giuliano A: Human papillomavirus infection in men attending a sexually transmitted disease clinic. J Infect Dis 2003;187:1064–1070.

Obstet Gynecol 1992;79:328–337. Bauer H, Hildesheim A, Schiffman M, Glass A, Rush B, Scott D, Cadell D, Kurman R, Manos M: Determinants of genital human papillomavirus infection in low-risk women in Portland, Oregon. Sex Transm Dis 1993;20:274–278. Nindl I, Lorincz A, Mielzynska I, Petry U, Baur S, Kirchmayr R , Michels W, Schneider A: Human papillomavirus detection in cervical intraepithelial neoplasia by the second-generation hybrid capture microplate test, comparing two different cervical specimen collection methods.

A non-intervention follow-up study of women with cytological evidence of low-grade cervical intra-epithelial neoplasia showed that HPV16 E2-specific T cell responses, as measured by specific IL-2 release in vitro, occurred frequently at the time of lesion clearance [26]. Good Th1-type immunity against the E2 and E6 protein has been detected in healthy individuals with no clinical signs of HPV16 infection [27, 28]. Importantly, these Th1-type responses were found only occasionally in high-grade cervical intra-epithelial neoplasia patients and were impaired in cervical cancer patients [29].

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